ras

Ras gene mutation in childhood AML was mainly found in M 2 , M 4 , M 5 subtypes.

AML患儿中ras基因突变多见于M2 、 M4及M5型.

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Conclusion HHS can inhibit the activation of local RAS in hemorrhagic shock.

结论　HHS可抑制失血性休克时组织中的RAS的激活.

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Objective : To investigate the correlation between RAS and SOD.

目的: 研究复发性口疮(RAS)与超氧化物歧化酶(SOD)活性的关系.

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C ras overexpression was associated with tumor differentiation.

高分化组的C-ras基因过度表达率显著高于低分化组.

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Objective : To approach the relationship between oncogene C - fos , K - ras and intrahep atic cholangiocarcinoma ( ICC ) and the significance.

目的: 探讨癌基因产物C -fos 、 K -ras 蛋白与肝内胆管癌发生的关系及其生物学意义.

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This article is intended to explain the RAS technique in FACOM M 340 computer system.

木文介绍了FACOMM340 中大型计算机系统设计中采用的RAS技术.

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Rapl, like Ras, cycles between a GDP bound inactive form and a GTP - bound active form.

Rap1,和Ras一样, 具有GDP结合的无活性形式和GTP结合的活性形式.

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The study aimed to analyze the effects of renin - angiotensin - system ( RAS ) blockade on renal endothelial function.

本研究旨在探索阻断肾素 - 血管紧张素系统 ( RAS ) 对肾脏内皮功能的影响.

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Cells with Ras gene mutation or transformed by Ras become resistant to p 53 dependent apoptosis.

Ras基因转染或突变的细胞能够对DNA损伤 引起的p53依赖性凋亡产生抗性.

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Continuing activation of ras gene will lead to cell proliferation and cause immortal cells.

ras基因的持续的激活可导致细胞的增殖和永生化.

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For instance, RAS cells developed into tumors when a wound was induced in the tissue.

例如当在组织中诱导创伤时可使RAS细胞发展成肿瘤.

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The circulating renin angiotensin system ( RAS ) is well maintenancepressure and electrolyte and fluid homeostasis.

除血循环外其他组织也存在一些局部肾素血管紧张素系统 ( RAS ) 调节机制.

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Objective : To confirm the cleavage activity of ribozyme Rz 199 to oncogene K ras messenger RNA.

目的: 确定人工设计的核酶Rz199对原癌基因K-rasmRNA的体外切割活性,为K-ras特异性核酶的体内应用提供依据.

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CC - Link fieldbus has higher performance in real - time characters, distributed control and RAS function etc.

CC-Link现场总线在 实时 性 、 分散控制、RAS功能等方面具有较高的性能.

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Conclusbo : It'suggsts that ras gene aed p 53 gene mutation may be related with L - form infedtbo.

结合提示L型感染参与了ras原癌基因激活和p53抑癌基因的失活.

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